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Items: 3

1.

Inactivation of CFTR by CRISPR/Cas9 alters transcriptional regulation of inflammatory pathways and other networks

(Submitter supplied) Individuals with cystic fibrosis (CF) experience elevated inflammation in multiple organs, but whether this reflects an inherent feature of CF cells or is a consequence of a pro-inflammatory environment is not clear. Using CRISPR/Cas9-mediated mutagenesis of CFTR, 17 subclonal cell lines were generated from Caco-2 cells. Clonal lines with functional CFTR (CFTR+) were compared to those without (CFTR-) to directly address the role of CFTR in inflammatory gene regulation. more...
Organism:
Homo sapiens
Type:
Expression profiling by high throughput sequencing
Platforms:
GPL16791 GPL21697
24 Samples
Download data: XLSX
Series
Accession:
GSE130226
ID:
200130226

PubMed Similar studies Analyze with GEO2RSRA Run Selector

2.

Illumina HiSeq 2500 (Homo sapiens)

Organism:
Homo sapiens
12216 Series
349569 Samples
Download data
Platform
Accession:
GPL16791
ID:
100016791

3.

CFTRneg_Treated_1

Organism:
Homo sapiens
Source name:
Caco2 cells
Platform:
GPL16791
Series:
GSE130226
Download data: XLSX
Sample
Accession:
GSM3734162
ID:
303734162

SRA Run Selector

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db=gds|term=GSM3734162[Accession]|query=1|qty=2|blobid=MCID_6748ccc943705129cf491edf|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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