Homo sapiens prominin 2 (PROM2), transcript variant 3, mRNA

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PROM2 overexpression induces metastatic potential through epithelial-to-mesenchymal transition and ferroptosis resistance in human cancers. [Clin Transl Med. 2024]
PROM2 overexpression induces metastatic potential through epithelial-to-mesenchymal transition and ferroptosis resistance in human cancers.
Paris J, Wilhelm C, Lebbé C, Elmallah M, Pamoukdjian F, Héraud A, Gapihan G, Walle AV, Tran VN, Hamdan D, et al. Clin Transl Med. 2024 Mar; 14(3):e1632.
Prominin 2 decreases cisplatin sensitivity in non-small cell lung cancer and is modulated by CTCC binding factor. [Radiol Oncol. 2023]
Prominin 2 decreases cisplatin sensitivity in non-small cell lung cancer and is modulated by CTCC binding factor.
Tang J, Shu D, Fang Z, Yang G. Radiol Oncol. 2023 Sep 1; 57(3):325-336. Epub 2023 Sep 4.
Elevation of hsa-miR-7-5p level mediated by CtBP1-p300-AP1 complex targets ATXN1 to trigger NF-κB-dependent inflammation response. [J Mol Med (Berl). 2023]
Elevation of hsa-miR-7-5p level mediated by CtBP1-p300-AP1 complex targets ATXN1 to trigger NF-κB-dependent inflammation response.
Lou LQ, Zhou WQ, Song X, Chen Z. J Mol Med (Berl). 2023 Mar; 101(3):223-235. Epub 2023 Jan 11.

RefSeq alternative splicing
See 13 reference mRNA sequence splice variants for the PROM2 gene.
RefSeq protein product
See the reference protein sequence for prominin-2 isoform 1 precursor (NP_653308.2).

Protein
Protein sequence from Nucleotide coding region
PubMed
PubMed articles cited by Nucleotide sequence record
Taxonomy
Taxonomy sequences associated with Nucleotide record
Annotated Genomic
Genomic sequence records that have the current RNA records as annotated features marking the exons of genes.
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CCDS
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Components (Core)
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Functional Class
Functional class of the sequence domain architecture
Gene
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OMIM
OMIM records associated with Nucleotide sequence
Probe
Related Probe entry
PubMed (RefSeq)
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PubMed (Weighted)
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GenScript latest version of gene cDNA ORF Clone [GenScript latest version of g...]
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Nucleotide