GSM60357[Accession] - GEO DataSets

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Items: 4

Select item 2000027701.

Transcriptional profiles of Th cells induced to polarize to Th1 or Th2 direction in the presence or absence of TGFbeta

(Submitter supplied) Th1 and Th2 cells arise from a common precursor cell in response to triggering through the TCR and cytokine receptors for IL-12 or IL-4. This leads to activation of complex signaling pathways, which are not known in detail. Disturbances in the balance between type 1 and type 2 responses can lead to certain immune-mediated diseases. Thus, it is important to understand how Th1 and Th2 cells are generated. more...

Organism:: Homo sapiens

Type:: Expression profiling by array

Dataset:: GDS1290
Platforms:: GPL97 GPL96 GPL8300
102 Samples

Download data: CEL

Series

Accession:: GSE2770

ID:: 200002770

Select item 12902.

CD4+ lymphocyte polarization into Th1 and Th2 cells in the presence of TGFbeta: time course (HG-U95A)

Analysis of CD4+ lymphocyte induced to differentiate into Th1 and Th2 by treatment with IL-12 and IL-4 respectively in the presence of TGFbeta. Cells examined at various time points up to 48 hours after treatment. Results provide insight into the mechanisms regulating CD4+ cell polarization.

Organism:: Homo sapiens

Type:: Expression profiling by array, count, 5 agent, 2 other, 4 time sets

Platform:: GPL8300
Series:: GSE2770
34 Samples

Download data: CEL

DataSet

Accession:: GDS1290

ID:: 1290

Select item 1000083003.

[HG_U95Av2] Affymetrix Human Genome U95 Version 2 Array

(Submitter supplied) Affymetrix submissions are typically submitted to GEO using the GEOarchive method described at http://www.ncbi.nlm.nih.gov/projects/geo/info/geo_affy.html June 03, 2009: annotation table updated with netaffx build 28 June 08, 2012: annotation table updated with netaffx build 32 June 27, 2016: annotation table updated with netaffx build 35 Protocol: see manufacturer's web site Based on this UniGene build and associated annotations, the HG-U95Av2 array represents approximately 10,000 full-length genes. more...