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Items: 3

1.

Inactivation of CFTR by CRISPR/Cas9 alters transcriptional regulation of inflammatory pathways and other networks

(Submitter supplied) Individuals with cystic fibrosis (CF) experience elevated inflammation in multiple organs, but whether this reflects an inherent feature of CF cells or is a consequence of a pro-inflammatory environment is not clear. Using CRISPR/Cas9-mediated mutagenesis of CFTR, 17 subclonal cell lines were generated from Caco-2 cells. Clonal lines with functional CFTR (CFTR+) were compared to those without (CFTR-) to directly address the role of CFTR in inflammatory gene regulation. more...
Organism:
Homo sapiens
Type:
Expression profiling by high throughput sequencing
Platforms:
GPL21697 GPL16791
24 Samples
Download data: XLSX
Series
Accession:
GSE130226
ID:
200130226

PubMed Similar studies Analyze with GEO2RSRA Run Selector

2.

NextSeq 550 (Homo sapiens)

Organism:
Homo sapiens
1500 Series
44597 Samples
Download data
Platform
Accession:
GPL21697
ID:
100021697

3.

CFTRpos_Nontreated_4

Organism:
Homo sapiens
Source name:
Caco2 cells
Platform:
GPL21697
Series:
GSE130226
Download data: XLSX
Sample
Accession:
GSM3734147
ID:
303734147

SRA Run Selector

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db=gds|term=GSM3734147[Accession]|query=1|qty=2|blobid=MCID_674c293796d9ad04062038c1|ismultiple=true|min_list=5|max_list=20|def_tree=20|def_list=|def_view=|url=/Taxonomy/backend/subset.cgi?|trace_url=/stat?
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